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This medicine is a selective serotonin reuptake inhibitor (SSRI) used to treat depression, panic disorder, obsessive-compulsive disorder (OCD), post-traumatic stress disorder (PTSD), social anxiety disorder (social phobia), and a severe form of premenstrual syndrome called premenstrual dysphoric disorder (PMDD).

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Zoloft o generico. Fluoxetine and olanzapine were not significantly associated with an increase or decrease of overall mortality. There was a significant interaction with gender on mortality rates, F 1,24 =8.88, p < 0.004. When the analysis was restricted to male individuals, Fluoxetine-treated males had 4.2 times (95% CI 2.4-5.7) the absolute rate of mortality, relative to control participants, when comparing the first 6 months of study, after adjustment for age, and prior disease severity. No other interactions were found, when stratifying by gender (Table 3). To examine the effect of fluoxetine on mortality rate during the first 6 months, it was necessary to examine mortality in the same time period for participants with no prior diagnosis of depression, and to evaluate the interaction with risk for a subsequent diagnosis. those participants with a prior diagnosis of depression, the hazard ratios overall mortality were similar to the unadjusted findings. risk of first 6 months follow-up was significantly attenuated (hazard ratio =0.84; 95% CI 0.70-1.01) for those who had a previous diagnosis of depression (p < 0.001). In the sub-analysis to determine impact of an increase in the risk for a subsequent diagnosis following fluoxetine treatment, the hazard ratios of mortality during the first 24 months of follow-up were no different from those when adjusted for covariates or non-fluoxetine therapies (Table 3). There was no significant impact of fluoxetine on the risk death due to pneumonia for participants not treated with olanzapine or fluoxetine (1.08% (1.00-1.19) for the total population, respectively). In this analysis, the hazard of mortality for fluoxetine-treated participants was also not significantly different from that of participants in the other two treatment groups (p = 0.24). Discussion The present study shows a trend towards decreased mortality over the first 6 months of fluoxetine treatment, but overall mortality rates remained substantially elevated throughout the study. This is consistent with other large studies (Hoffman et al., 2002; Rabinovich 2003; Fusar-Poli et al., 2004; Nader 2005). However, the effect of fluoxetine on mortality rates was less pronounced in the subset with a prior diagnosis of depression than in those without. This may reflect the fact that individuals with a prior diagnosis of depression had a significantly higher initial risk of suicide, and therefore a lower initial baseline incidence of fluoxetine-associated mortality than non-depressed participants. This is in contrast to other studies which this association was not observed (e.g. Hoffman et al., 2002; Rabinovich 2003; Fusar-Poli et al., 2004; Nader 2005a). In the presence of a previous diagnosis depression, fluoxetine treatment significantly decreased mortality in the first 12 months of follow up. This may be explained by fluoxetine-associated symptom reduction, with the risk being Zoloft 90 Pills 50mg $95 - $1.06 Per pill lower in first several months following treatment. In a recent study, the overall survival rate was similar at 6 and 24 months for those patients on fluoxetine (Eddy et al., 2005). Thus, we did not find that improvement in symptoms was the main effect for improving subsequent mortality, which may be more a reflection of delay in the onset depression. In a recent study, which similar number of participants with suicide risk were studied compared with a comparison cohort of depressed patients with a prior diagnosis of depression, the overall survival depressed patients on treatment was higher at 6 months than 24 (Gottlieb et al., 2004). In this study, a decrease the rate of suicide was related to symptom reduction rather than an increase in overall survival rate. There was also no statistically significant difference between symptom improvements in the first 24 months of follow-up for the depressed population with a prior diagnosis of depression and the non-depressed population. Other studies have not shown that depression is a risk factor for mortality (Nader et al., 2005b). The study by Eddy et al., however, did show a tendency towards worsening of symptoms in the first year following antidepressant treatment in suicidal patients with a prior diagnosis of depression at baseline and a reduction of the rate suicide during 12 months following treatment. This is consistent with other studies, in which improvement symptoms was a consistent and significant association for reduction in mortality (e.g. Fusar-Poli et al., 2004), and that depressive symptoms also had a direct effect on both overall survival and mortality. In the present study, there was no significant interaction between baseline diagnosis of depression and mortality fluoxet.



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